Erythema multiforme - Érythema Multiforme
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Erythema multiforme minor ― Catet yén puseur lesi tiasa blanch.
Urtikaria ogé tiasa dianggap salaku diagnosis diferensial.
relevance score : -100.0%
ReferencesErythema multiforme (EM) mangrupikeun kaayaan dimana bintik‑bintik anu siga target muncul dina kulit sareng mémbran mukosa kusabab réaksi imun. Sanajan mindeng dipicu ku inféksi viral, utamana virus herpes simpleks (HSV), atawa pangobatan tangtu, sababna masih teu dipikanyaho dina loba kasus. Ngubaran EM akut museurkeun kana ngaleungitkeun gejala ku ngagunakeun krim anu ngandung steroid atanapi antihistamin. Ngatur EM anu kambuh paling éféktif lamun disaluyukeun ka unggal pasien. Pendekatan awal ngalibatkeun perlakuan lisan sareng topikal, kalebet kortikosteroid sareng pangobatan antiviral. Pangobatan topikal diwangun ku krim steroid anu kuat sareng solusi pikeun mémbran mukosa anu kapangaruhan. Pikeun pasien anu henteu ngaréspon kana antiviral, pilihan jalur kadua kalebet ubar anu ngahambat sistem kekebalan, antibiotik, anthelmintik, sareng antimalarial.
Erythema multiforme (EM) is an immune-mediated condition that classically presents with discrete targetoid lesions and can involve both mucosal and cutaneous sites. While EM is typically preceded by viral infections, most notably herpes simplex virus (HSV), and certain medications, a large portion of cases are due to an unidentifiable cause. Treatment for acute EM is focused on relieving symptoms with topical steroids or antihistamines. Treatment for recurrent EM is most successful when tailored to individual patients. First line treatment for recurrent EM includes both systemic and topical therapies. Systemic therapies include corticosteroid therapy and antiviral prophylaxis. Topical therapies include high-potency corticosteroids, and antiseptic or anesthetic solutions for mucosal involvement. Second-line therapies for patients who do not respond to antiviral medications include immunosuppressive agents, antibiotics, anthelmintics, and antimalarials
Erythema multiforme (EM) is an immune-mediated condition that classically presents with discrete targetoid lesions and can involve both mucosal and cutaneous sites. While EM is typically preceded by viral infections, most notably herpes simplex virus (HSV), and certain medications, a large portion of cases are due to an unidentifiable cause. Treatment for acute EM is focused on relieving symptoms with topical steroids or antihistamines. Treatment for recurrent EM is most successful when tailored to individual patients. First line treatment for recurrent EM includes both systemic and topical therapies. Systemic therapies include corticosteroid therapy and antiviral prophylaxis. Topical therapies include high-potency corticosteroids, and antiseptic or anesthetic solutions for mucosal involvement. Second-line therapies for patients who do not respond to antiviral medications include immunosuppressive agents, antibiotics, anthelmintics, and antimalarials
Dina sababaraha kasus, erythema multiforme hampang leungit sorangan dina 2 nepi ka 4 minggu. Sindrom Stevens-Johnson, kaayaan parna anu mangaruhan mémbran mukosa, tiasa dugi ka 6 minggu. Steroid biasana henteu disarankeun pikeun kasus anu hampang. Naha steroid kudu dipaké pikeun erythema multiforme parna teu pasti, sabab euweuh papanggihan jelas tina studi randomized nunjukkeun barudak bakal nguntungkeun tina perlakuan ieu.
In most cases, mild erythema multiforme is self-limited and resolves in 2 to 4 weeks. Stevens-Johnson syndrome is a serious disease that involves the mucous membranes and lasts up to 6 weeks. There is no indication for using steroids for the mild form. Use of steroids for erythema multiforme major is debatable because no randomized studies clearly indicate which children will benefit from this treatment.
In most cases, mild erythema multiforme is self-limited and resolves in 2 to 4 weeks. Stevens-Johnson syndrome is a serious disease that involves the mucous membranes and lasts up to 6 weeks. There is no indication for using steroids for the mild form. Use of steroids for erythema multiforme major is debatable because no randomized studies clearly indicate which children will benefit from this treatment.
Kami nampilkeun kasus erythema multiforme (EM) lisan anu disababkeun ku TMP/SMX, némbongkeun borok lisan jeung bibir tanpa lesi kulit. Ieu negeskeun kabutuhan pikeun ngabédakeunana tina gangguan ulcerative lisan anu sanés. Pasien nampi perlakuan simptomatik sarta tablet prednisolone, ngahontal perbaikan sanggeus ngeureunkeun terapi TMP/SMX.
We report a case of oral erythema multiforme (EM) secondary to TMP/SMX that presented with oral and lip ulcerations typical of EM without any skin lesions and highlights the importance of distinguishing them from other ulcerative disorders involving oral cavity. The patient was treated symptomatically and given tablet prednisolone. The condition improved with stoppage of TMP/SMX therapy.
We report a case of oral erythema multiforme (EM) secondary to TMP/SMX that presented with oral and lip ulcerations typical of EM without any skin lesions and highlights the importance of distinguishing them from other ulcerative disorders involving oral cavity. The patient was treated symptomatically and given tablet prednisolone. The condition improved with stoppage of TMP/SMX therapy.
Erythema multiforme mangrupakeun réaksi nu ngalibetkeun kulit sarta kadangkala mukosa, dipicu ku sistim imun. Ilaharna, éta muncul salaku lesi target‑kawas, nu bisa muncul sorangan, kambuh, atawa tetep. Lesi ieu biasana mangaruhan sacara simetris kana ekstremitas, hususna dina permukaan luar. Panyabab utama kalebet inféksi sapertos virus herpes simplex jeung Mycoplasma pneumoniae, ogé pangobatan, imunisasi, jeung panyakit otoimun. Ngabédakeun erythema multiforme ti urtikaria gumantung kana lilana lesi; lesi erythema multiforme tetep sahenteuna tujuh poé, sedengkeun lesi urtikaria sering ngaleungit dina hiji poé. Sanaos sarupa, penting pisan pikeun ngabédakeun erythema multiforme tina sindrom Stevens‑Johnson anu leuwih parah, anu biasana nembongkeun macula erythematous atawa purpuric jeung lepuh. Ngatur erythema multiforme ngalibatkeun ngaleungitkeun gejala ku kortikosteroid topikal atawa antihistamin sarta ngungkulan panyababna. Pikeun kasus anu kambuh patali jeung virus herpes simplex, terapi antiviral profilaksis disarankeun. Keterlibatan mukosa anu parah tiasa merlukeun rawat inap pikeun cairan intravena jeung ngagantian éléktrolit.
Erythema multiforme is a reaction involving the skin and sometimes the mucosa, triggered by the immune system. Typically, it manifests as target-like lesions, which may appear isolated, recur, or persist. These lesions usually symmetrically affect the extremities, particularly their outer surfaces. The main causes include infections like herpes simplex virus and Mycoplasma pneumoniae, as well as certain medications, immunizations, and autoimmune diseases. Distinguishing erythema multiforme from urticaria relies on the duration of lesions; erythema multiforme lesions remain fixed for at least seven days, while urticarial lesions often vanish within a day. Although similar, it's crucial to differentiate erythema multiforme from the more severe Stevens-Johnson syndrome, which typically presents widespread erythematous or purpuric macules with blisters. Managing erythema multiforme involves symptomatic relief with topical steroids or antihistamines and addressing the underlying cause. For recurrent cases associated with herpes simplex virus, prophylactic antiviral therapy is recommended. Severe mucosal involvement may necessitate hospitalization for intravenous fluids and electrolyte replacement.
Erythema multiforme is a reaction involving the skin and sometimes the mucosa, triggered by the immune system. Typically, it manifests as target-like lesions, which may appear isolated, recur, or persist. These lesions usually symmetrically affect the extremities, particularly their outer surfaces. The main causes include infections like herpes simplex virus and Mycoplasma pneumoniae, as well as certain medications, immunizations, and autoimmune diseases. Distinguishing erythema multiforme from urticaria relies on the duration of lesions; erythema multiforme lesions remain fixed for at least seven days, while urticarial lesions often vanish within a day. Although similar, it's crucial to differentiate erythema multiforme from the more severe Stevens-Johnson syndrome, which typically presents widespread erythematous or purpuric macules with blisters. Managing erythema multiforme involves symptomatic relief with topical steroids or antihistamines and addressing the underlying cause. For recurrent cases associated with herpes simplex virus, prophylactic antiviral therapy is recommended. Severe mucosal involvement may necessitate hospitalization for intravenous fluids and electrolyte replacement.
Kaayaanana rupa‑rupa, ti nu hampang nepi ka nu parah, kalebet bentuk anu ngancam kahirupan nu disebut erythema multiforme major anu ogé ngalibatkeun mémbran mukosa. Invasi mémbran mukosa atawa ayana bula mangrupa tanda parah.
- Erythema multiforme minor: lesi target atawa diangkat, papul edema sumebar sacara akral.
Bentuk hampang biasana muncul kalayan rasa gatel (sanajan gatelna tiasa parah), bintik‑bintik pink‑beureum nu simetris jeung dimimitian ti ujung‑ujung. Penyembuhan biasana lumangsung dina 7‑10 poé.
- Erythema multiforme major: lesi target atawa diangkat, papul edema sumebar sacara akral kalawan keterlibatan hiji atawa leuwih mémbran mukosa. Detasemen épidermis ngalibatkeun kirang ti 10 % total luas permukaan awak.
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